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At some point should mention the Brockenbrough-Braunwald-Morrow Sign, which is a decrease in the pulse pressure after a premature ventricular contraction. While the left ventricular pressure increases in the beat after the premature contraction, the dynamic obstruction increases to a greater degree, so that the arterial pressure paradoxically decreases. An individual with a fixed obstruction (for instance, due to
aortic stenosis), the degree of obstruction will not change, and the left ventricular and systemic systolic pressures will both increase in the beat after the premature ventricular complex.
Ksheka 02:59, Aug 25, 2004 (UTC)
This is something to watch: soy diet worsened HCM risks in mice. [1]. JFW | T@lk 00:04, 5 January 2006 (UTC)
Where did Ulrich Sigwart report his first case series with septal ablation? JFW | T@lk 22:22, 1 February 2006 (UTC)
At regular times a professional bodybuilder dies. Afterwards in the autopsy doctors often find a hypertrophied heart. Is there any connection between steroid usage and HC? Sijo Ripa 19:08, 23 July 2006 (UTC)
Information is needed on echocardiography, Cardiac MRI and ECG in regards to diagnostic testing. — 62.6.139.11 comment added 14:06, 10 October 2006
Broadening the indication for ICDs: JAMA JFW | T@lk 23:23, 24 July 2007 (UTC)
I had Ripley's Believe it or Not on it they showed a woman with this who's symptoms were cured or severely reduced by a forced heart attack. I believe this should be mentioned in the article, though I'm not sure if it has become a possible solution for most, or if this is the only case.
I had First Respone at my house because a friend was freakinjg out and thought he had it. There computers andthe person that came over said it doesnt exist???
The origin of the hypothesis, that systolic anterior motion of the anterior leaflet of the mitral valve is caused by a venturi effect, appears to be Wigle E. D., Adelman A. G., Silver M. D.: Pathophysiological considerations in muscular subaortic stenosis. In Wolstenholme G. E. W, O'Connor M., editors: Hypertrophic obstructive cardiomyopathy. London, 1971, Ciba Foundation Study Group, No. 47, Churchill, p. 63.
Evidence of this that is available on-line ( http://circ.ahajournals.org/cgi/reprint/75/2/311.pdf) as of 1/18/10 is the following editorial in Circulation. E. D. Wigle, Hypertrophic cardiomyopathy: a 1987 viewpoint. Circulation (1987) 75: 311-322. There, Dr. Wigle claims the hypothesis on page 312, and the above reference is citation 11.
A contributor having access to the original article might choose to include it as the missing reference in this Wikipedia article. Nhy67ygv ( talk) 06:50, 19 January 2010 (UTC)
This merger makes a lot of sense since the article on Hypertrophic Cardiomyopathy Screening is an orphan page, is not too long, and is relevant to people looking up Hypertrophic Cardiomyopathy. Screening is more of a section than a standalone article. Gccwang ( talk) 07:15, 19 January 2010 (UTC)
From treatment section:"A high risk of suffering sudden cardiac death may be indicated by a ventricular wall thickness greater than 30mm, unexplained syncope, family history of sudden cardiac death, abnormal blood pressures after exercise.." What is a normal blood pressure after exercise. Does one's BP normally go up or does it normally go down after exercis? 76.218.104.120 ( talk) 21:19, 18 June 2012 (UTC)
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This page is an archive of past discussions. Do not edit the contents of this page. If you wish to start a new discussion or revive an old one, please do so on the current talk page. |