Pemphigus erythematosus patients typically present with superficially eroded lesions, or vesiculobullae, that may ooze and crust. This is especially common in areas of the body that are exposed to the sun, like the back, upper chest, and face.[3]
The symptoms of pemphigus erythematosus usually appear slowly and progress slowly. The patient might not even be aware that their pemphigus erythematosus is photosensitive, even though the distribution of the lesions should indicate that they were induced by sunlight.[3]
Causes
Pemphigus patients experience an autoimmune reaction that targets
desmosomes.[4] pemphigus erythematosus relapse has been linked to
atorvastatin use.[5] There has been one report of a new case of pemphigus erythematosus following topical
ingenol mebutate treatment.[6]
^Oktarina, Dyah A. M.; Poot, Angelique M.; Kramer, Duco; Diercks, Gilles F. H.; Jonkman, Marcel F.; Pas, Hendri H. (October 1, 2012). "The IgG "Lupus-Band" Deposition Pattern of Pemphigus Erythematosus". Archives of Dermatology. 148 (10). American Medical Association (AMA): 1173–1178.
doi:
10.1001/archdermatol.2012.1896.
ISSN0003-987X.
PMID22801864.
Amerian, Mary L.; Ahmed, A. Razzaque (1984). "Pemphigus erythematosus". Journal of the American Academy of Dermatology. 10 (2). Elsevier BV: 215–222.
doi:
10.1016/s0190-9622(84)70025-9.
ISSN0190-9622.